Targeting CSC Dormancy
Cancer stem cells (CSCs) can enter a dormant state, allowing them to evade treatments and later reinitiate tumor growth. This track focuses on understanding the mechanisms that regulate CSC dormancy, including signals from the microenvironment and intrinsic genetic factors. Researchers explore how dormant CSCs resist conventional therapies, which typically target actively dividing cells. Developing therapies that can either awaken dormant CSCs, making them vulnerable to treatment, or eliminate them while dormant, could significantly improve long-term survival rates for cancer patients. This research aims to prevent tumor recurrence by targeting the CSCs that remain dormant but capable of causing relapse.
Related Conference of Targeting CSC Dormancy
November 25-26, 2024
7th International Conference on Anti-Cancer Drugs & Therapies
Vancouver, Canada
April 21-22, 2025
16th International Conference on Cancer Stem Cell & Oncology Research
Amsterdam, Netherlands
Targeting CSC Dormancy Conference Speakers
Recommended Sessions
- CRISPR/Cas9 in CSC Research
- CSC and Immunoediting
- CSC Biomarkers for Early Detection
- CSC Fate Determination and Differentiation
- CSC Heterogeneity in Tumors
- CSC Metabolism and Energy Pathways
- CSC Niche and Hypoxia
- CSC Plasticity and Tumor Progression
- CSC-Derived Exosomes and Communication
- CSC-Driven Angiogenesis
- CSC-Driven Resistance to Radiotherapy
- CSC-Mediated Immunosuppression
- CSCs and Tumor Microenvironment
- Drug Resistance Mechanisms in CSCs
- Epigenetics and CSC Regulation
- Immunotherapy and CSCs
- Inflammation and CSC Activation
- Molecular Signaling in CSCs
- Nanotechnology and CSC Targeting
- Natural Products and CSC Targeting
- Role of CSCs in Metastasis
- Role of EMT in CSC Plasticity
- Targeting CSC Dormancy
- Targeting CSC-Specific Surface Markers
- Therapy Resistance and CSCs
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