Inflammation and CSC Activation
Chronic inflammation plays a significant role in cancer initiation and progression, often leading to the activation of cancer stem cells (CSCs). This track investigates the links between inflammatory signaling pathways—such as NF-κB, IL-6, and TNF-α—and the activation of CSCs. Researchers explore how inflammation within the tumor microenvironment promotes CSC self-renewal, survival, and metastasis. Targeting the inflammatory signals that support CSCs could lead to novel therapeutic strategies that disrupt this relationship, limiting tumor growth and progression. This research highlights the importance of anti-inflammatory agents in cancer treatment, offering new opportunities to inhibit CSC-driven malignancies by reducing inflammation.
Related Conference of Inflammation and CSC Activation
April 21-22, 2025
16th International Conference on Cancer Stem Cell & Oncology Research
Amsterdam, Netherlands
October 13-14, 2025
8th International Conference on Anti-Cancer Drugs & Therapies
Aix-en-Provence, France
November 24-25, 2025
47th Euro-Global Summit on Cancer Therapy & Radiation Oncology
Barcelona, Spain
Inflammation and CSC Activation Conference Speakers
Recommended Sessions
- CRISPR/Cas9 in CSC Research
- CSC and Immunoediting
- CSC Biomarkers for Early Detection
- CSC Fate Determination and Differentiation
- CSC Heterogeneity in Tumors
- CSC Metabolism and Energy Pathways
- CSC Niche and Hypoxia
- CSC Plasticity and Tumor Progression
- CSC-Derived Exosomes and Communication
- CSC-Driven Angiogenesis
- CSC-Driven Resistance to Radiotherapy
- CSC-Mediated Immunosuppression
- CSCs and Tumor Microenvironment
- Drug Resistance Mechanisms in CSCs
- Epigenetics and CSC Regulation
- Immunotherapy and CSCs
- Inflammation and CSC Activation
- Molecular Signaling in CSCs
- Nanotechnology and CSC Targeting
- Natural Products and CSC Targeting
- Role of CSCs in Metastasis
- Role of EMT in CSC Plasticity
- Targeting CSC Dormancy
- Targeting CSC-Specific Surface Markers
- Therapy Resistance and CSCs
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