CSC Fate Determination and Differentiation
Cancer stem cells (CSCs) possess the unique ability to self-renew and differentiate into various cell types within the tumor. This track explores the molecular mechanisms that govern CSC fate determination, including how signals from the tumor microenvironment and intrinsic genetic factors influence whether a CSC remains stem-like or differentiates. Researchers study how manipulating CSC differentiation pathways could force CSCs to lose their stemness and become more susceptible to treatment. By inducing CSC differentiation, therapies could target the more differentiated, less resistant cells, potentially preventing tumor growth and recurrence. This research aims to develop new strategies that alter CSC fate and improve cancer therapy outcomes.
Related Conference of CSC Fate Determination and Differentiation
April 21-22, 2025
16th International Conference on Cancer Stem Cell & Oncology Research
Amsterdam, Netherlands
November 24-25, 2025
47th Euro-Global Summit on Cancer Therapy & Radiation Oncology
Barcelona, Spain
CSC Fate Determination and Differentiation Conference Speakers
Recommended Sessions
- CRISPR/Cas9 in CSC Research
- CSC and Immunoediting
- CSC Biomarkers for Early Detection
- CSC Fate Determination and Differentiation
- CSC Heterogeneity in Tumors
- CSC Metabolism and Energy Pathways
- CSC Niche and Hypoxia
- CSC Plasticity and Tumor Progression
- CSC-Derived Exosomes and Communication
- CSC-Driven Angiogenesis
- CSC-Driven Resistance to Radiotherapy
- CSC-Mediated Immunosuppression
- CSCs and Tumor Microenvironment
- Drug Resistance Mechanisms in CSCs
- Epigenetics and CSC Regulation
- Immunotherapy and CSCs
- Inflammation and CSC Activation
- Molecular Signaling in CSCs
- Nanotechnology and CSC Targeting
- Natural Products and CSC Targeting
- Role of CSCs in Metastasis
- Role of EMT in CSC Plasticity
- Targeting CSC Dormancy
- Targeting CSC-Specific Surface Markers
- Therapy Resistance and CSCs
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