CSC Plasticity and Tumor Progression
Cancer stem cells (CSCs) exhibit remarkable plasticity, allowing them to switch between stem-like and differentiated states in response to microenvironmental cues. This track explores the mechanisms underlying CSC plasticity and how it contributes to tumor progression, metastasis, and therapeutic resistance. Researchers study the factors that regulate transitions between CSC and non-CSC states, including epigenetic changes and signaling pathways. Targeting CSC plasticity could prevent tumors from adapting to therapies and evolving into more aggressive forms. By understanding how plasticity drives tumor heterogeneity and survival, researchers aim to develop therapies that eliminate CSCs and block tumor progression, reducing the chances of relapse.
Related Conference of CSC Plasticity and Tumor Progression
November 25-26, 2024
7th International Conference on Anti-Cancer Drugs & Therapies
Vancouver, Canada
April 21-22, 2025
16th International Conference on Cancer Stem Cell & Oncology Research
Amsterdam, Netherlands
CSC Plasticity and Tumor Progression Conference Speakers
Recommended Sessions
- CRISPR/Cas9 in CSC Research
- CSC and Immunoediting
- CSC Biomarkers for Early Detection
- CSC Fate Determination and Differentiation
- CSC Heterogeneity in Tumors
- CSC Metabolism and Energy Pathways
- CSC Niche and Hypoxia
- CSC Plasticity and Tumor Progression
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- CSC-Driven Angiogenesis
- CSC-Driven Resistance to Radiotherapy
- CSC-Mediated Immunosuppression
- CSCs and Tumor Microenvironment
- Drug Resistance Mechanisms in CSCs
- Epigenetics and CSC Regulation
- Immunotherapy and CSCs
- Inflammation and CSC Activation
- Molecular Signaling in CSCs
- Nanotechnology and CSC Targeting
- Natural Products and CSC Targeting
- Role of CSCs in Metastasis
- Role of EMT in CSC Plasticity
- Targeting CSC Dormancy
- Targeting CSC-Specific Surface Markers
- Therapy Resistance and CSCs
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